Endocrine Disruptors
Endocrine-disrupting chemicals may contribute to obesity with growing epidemiological and mechanistic evidence. The session covers BPA, phthalates, and PFAS evidence in obesity, microplastics and metabolic health, dietary-exposure routes, regulatory frameworks (FDA, EFSA), emerging epidemiological evidence including the EARTH and CHAMACOS cohorts. Discussion addresses the obesogen hypothesis, gestational exposure and offspring obesity, the role of EDCs in PCOS and male hypogonadism, occupational exposures, the policy levers including chemical regulation, the comparison of conventional vs organic diets in EDC exposure, and the integration of environmental medicine in obesity practice.
- BPA and phthalates evidence
- PFAS and obesity
- Microplastics and metabolic health
- Dietary exposure routes
- EARTH and CHAMACOS cohorts
- Obesogen hypothesis
- Gestational EDC exposure
- Environmental medicine integration
Explore the full GSOD 2027 program
- 01GLP-1 & Incretin Therapies
- 02Bariatric Surgery
- 03Childhood & Adolescent Obesity
- 04Obesity Genetics & Epigenetics
- 05Metabolic Syndrome
- 06Behavioral & Lifestyle Interventions
- 07Gut Microbiome & Metabolism
- 08Health Equity in Obesity Care
- 09Policy, Environment & Prevention
- 10Weight Loss Maintenance
- 11Eating Disorders & Obesity
- 12Sarcopenic Obesity
- 13Hormonal Drivers
- 14Cardiometabolic Complications
- 15MASH & Obesity-Liver Axis
- 16Sleep & Obesity
- 17Workplace & School Programs
- 18Pharmacotherapy Pipeline
- 19Type 1 Diabetes
- 20Type 2 Diabetes
- 21Diabetic Retinopathy
- 22Diabetic Neuropathy
- 23Diabetic Kidney Disease
- 24Polycystic Ovary Syndrome
- 25Thyroid & Weight
- 26Insulin Resistance
- 27Lipid Metabolism
- 28Body Composition
- 29Body Image & Mental Health
- 30Diabetes in Pregnancy
- 31Telemedicine in Obesity
- 33Health Coaching
- 34Geriatric Obesity
- 35Stigma & Communication